Introduction
Atherosclerosis is a continuum of lesions caused by the deposits of cholesterol in the arterial wall, which is favored by the circulating oxidized low density lipoprotein (LDL) cholesterol (Adelmann, 2011). The genetic predisposition to atherosclerosis is associated with specific genes or certain gene variants. The platelet glycoprotein IIIa is viewed to be a prothrombotic predisposing factor among those with a family history of premature myocardial infarction. This genetic predisposition to atherosclerosis appears to interact with a cholesterol level (Shoenfeld, Harats and Wick, 2001). Genetic disorders involving a low, high density lipoprotein (HDL) cholesterol are also associated with an increased Continue reading...